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STUDY ON PRODUCTION PERFORMANCE OF BROILER PARENT STOCK (COBB-500) IN NAHAR POULTRY FARM AT MIRAISARI, CHITTAGONG

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ABSTRACT

The study was conducted at Chittagong Veterinary and Animal Sciences University ,aiming to know, the cause of dystocia due to excessive fluid loss . For this purpose, the history of this goat was recorded. Case finding revealed that 3 years old Blake Bengal goat was presented at SA quaderi teaching veterinary hospital,CVASU with the history of profuse fluid discharge through vagina ,. On the basis of the clinical history,it was diagnosed as dystocia. Then still birth of two death fetus. The fetus was very large in size and second was larger than first one. Then dam treated with oxytocin,decam and 5% DNS.

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INTRODUCTION

The term dystocia comes from a greek word means difficult birth. When the 1st or specially the second stage of parturition is markedly prolonged or becomes difficult or impossible for the dam to deliver without artificial aid this condition is termed as dystocia(Bellowas RA.1996). Its diagnosis and treatment constitute a large and important part of science of obstetrics (Arthur et al 1996). Kidding difficulty contributes heavily to production losses in kid herds. The losses are due to kids that die at or soon after kidding(Majeed,A.F.1994) .. Dystocia may result in reduced kid performance, delayed estrus and in some cases, Loss of the kid. The majority of non disease related kid losses in herds consist of kid death associated with dystocia.

Researchers from Montana evaluated 798 autopsied kids lost in disease free herd over a 15 year period. They determined that more than twice as many kids are lost around the time of kidding than at any other time from birth to wearing. .

difficult birth is a contributory factor in peri-natal death of dams and newborns because of damage to the birth canal and use of excessive traction force (Roke et al., 1990; Ghosh et al., 2004; Scott, 2005). The ability to distinguishing lambing and kidding difficulties is important in treating dystocia. Generally, dystocia may be of fetal or maternal origin (Noakes et al., 2009) Fetal dystocia occurred mainly due to oversize, mal-disposition, and monsters( Rahim and Majeed and Taha, 1989; Noakes et al., 2009). Maternal dystocia were mainly due to a deficient dilatation of the cervical canal (ringwomb), narrow pelvis and uterine inertia (Franklin, 1986; Majeed and Taha, 1989b; Thomas, 1992;Noakes et al., 2009 )

However, even large kids problems because they have never given birth. Buck kids, being langer, cause more problem. Kidding in winter are more likely to experience dystocia than those kidding in summer, probably because of exercise.

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As dystocia is usually considered a major cause of newborns and dams deaths with consequence severe economic losses thus the aims of this study were to describe the common causes of dystocia in Black Bengle goat in Bangladesh.

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REVIEW OF LITERATURE

Laster et all (1973) found that dam with had calving difficulties experience delay in resuming oestrus & showed 15.9% reduction in conception rate compared with cows which had calved normally.

Tutt (1944) has recorded 85.5% of fetal dystocia and 14.5% Due to maternal fraction.

Edwards (1979) recorded 66.5, 23.2& 14.3% of assisted deliveries in the first secon &

third calving in a Friesian herd.

Monton & cox (1968) found the three most important causes of dystocia to be fetal malpresentation 44.5%, fetomaternal disproportion 21.8%, uterine inertia 18%.

Vandenbussche et al(2001) reported that 50 percent of oversized fetuses in Belgium causing dystocia were double muschled.

In 800 birth Holsteins reported by Ben-David, 4.5% were posterior presentation but 47.2 percent of these were accompanied by dystocia.

By far the most common cause of dystocia is a kid larger than the birth canal. It means that either the birth canal of the dam is too small, quite a variety of factors may contribute to the small size of the dam & to the large size of the fetus (Benesch et al., 1951).

Scar tissue structure or fibrous adhesions which may be remnants of the healing of severe inflammation or traumatic injury from previous kidding may narrow all of parts of the birth canal. There may also be deformities, fractures, or dislocation of pelvic bones, which can decreses the size of pelvic inlet and cause dystocia. Sire

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selection can also play a role in the future cause of kidding. Unfortunately, these small dams have small birth canals which can not accommodate the huge kids. (Cunn, 1968) there are several abnormal conditions which produce oversized calves and thus dystocia. some of this conditions which produce oversized calves and thus dystocia.

Some of this conditions like rigid or contorted joints, hydrocephalus (enlarged, fluid filled head) and fused twins have genetic causes.

Fetal death can lead to a dystocia because the fetus plays a critical role in parturition.

Umbilical cord rupture or compression or trauma to the fetus can cause an enlarged fetus because of improper circulation and accumulation of blood and fluids within certain parts of the fetus. the fetus may bloat or become distended with gas after death and the increased size may prevent its easy delivery (wiltbank et al. ,1969)

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METHODOLOGY

The study was conducted to the SAQTVH. Reported animal is a local Black Bengal goat (age 3 years) and parity was one. Animal was housed under free condition

.Clinical history of this animal was bellowing from previous day but fetus was not expelled. Also additional history is that since one day, the water-bag of this goat was ruptured and all amniotic fluid was expelled. Clinical reported that visible mucous membrane was pink, dehydration, posture and gait is normal, vulvar discharge contain necrotic mass. The fetus become dry and fetus was large size. Inspite of this condition, reported case are treated by close observation of suspected problem. At first hands and instruments should be soaked in dilute iodine to prevent contamination from environment.Two dead fetus was removed manually.Then 2 ml oxytocin was injected i/m ly for hastens uterine involution and removed placenta.Oxytocin also helps to uterine contraction. Then 20 cc calcium preparation like decam injected i/v ly.Calcium act as synergestic action with oxytocin. Finally animal must receive adequate fluid therapy like 5% dextrose saline i/v ly.

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DISCUSSION

In case of SAQTVH hospital as the place in the metro politon area, people not rear so much doe in and around the hospital area. Basically large animal case is few, comparatively than small animal and that’s why dystocia case is very minimum. But many semi urban and urban area, people are aware about the animal, so if they face the problem, they take the patient in to the hospital and moreover large animal patient is more in rural and semi rural veterinary hospital and the incidence of the dystocia cases was 5%.

My report case is also a dystocia case. Reported animal is local goat, 3 years age and parity is one clinical history of this animal is bleating from one day but does not remove fetus. Also additional history is that since one day the water bag of this goat was ruptured and all water was expelled. Clinical examination reported that visible mucous membrane is pink, Dehydration, posture and gait is normal, vulvar discharge contain necrotic mass. Also clinical report that the fetus become dry and very large size.

By for the most common cause of dystocia is a kid larger than the birth canal. There are several abnormal conditions which produce oversized kids, Hydrocephalus (enlarged, fluid filled head) is one of them. As a result improper circulation and accumulation of blood and fluid may lead fetal dead and excessive fluid may expelled through vagina.

Reported case (excessive fluid loss causes dystocia) may suspect that displacement of the uterus occurs when the anterior part of the pregnant uterine horn which has grow out beyond its ligament, attachment rolls up over the small non pregnant horn, putting a giant twist in its longitudinal axis. Lock of exercise, weak abdominal muscles or anything increasing the mobility of the utrus may cause rupture the water bag, as a result loss of excessive fluid. On the other hand, falling from sloping place or any

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other rough handling during delivery, also cause for excessive fluid loss. As a result, low level of fluid within the uterus which puts the moving fetus in close contact with the uterine wall, may cause dystocia.

The proper use of resources requires an evaluation of the effects and cost of any medical action. This is particularly True for excessive fluid loss cause dystocia, where the treatment has typically applied almost exclusively. For the purpose to treated the goat, at first close observation of suspected problems and provide sanitation and proper area for kidding. Then hands instruments should be soaked in dilute iodine to prevent contamination of the birth canal since any day before the water gab of reported animal was ruptured and all water was expelled. So, the fetus become dry and fetus also very large in size. Inspite of this condition two dead fetus was removed by manually but the dam was ali

CONCLUSION

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Forms of dystocia in reported goat is very rare case. Fetal oversize were the most important causes of dystocia other less important causes were uterine torsion, as a result excessive fluid loss cause dystocia. Dystocia have great economic value in perspective of Bangladesh. The majority of non disease related kids losses, associated with dystocia. Not only in Bangladesh dystocia a great reproductive problem all over the world. Because it causes the loss of dam, fetus or both and also cause further complication eg pyometra, Retained placenta Endometritis, vaginal prolapsed uterine prolapsed etc.

REFERENCES

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Arthur et al.,1996,

Batista, M. Nitio, T, Alamo, D. Gonzalez F. Santana, M. Rodriguez, N. Cabrera, F. Gracia A. 2009 Use of Luprostiol and Cloprostenol for induction of parturition in pregnant goats. Reprod. Domest. Amin. 44, 83-87.

Bellowas RA. 1996

Brounts, S.H. Hawking, J.F. Bair. A.N. Glickman, L.T. 2004 Outcome and subsequent fertility of sheep and goats unergoing cesarean section because of dystocia:

110 cases (1981-2001). J. Am. Vet. Med. Assoc. 224, 275-279.

Dennis, S.M and. H.W. Leipold, 1979, Ovine Congenital Defects. The Veterinay Bulletin 49, 233-237.

Engum. J. Lyngset, O. 1970. Gynaecology and obstetrics in the goat. Iowa State Univ.

Vet. 120-124.

FAO. 2010 Intergoernmental Technical Working Gruoup on Animal Genetic Resources, Sixth Session, Rome, 24-26 November

Franklin, J.S. 1986. Dystocia and Obstetrics in Goats. In D.A. Morrow (Editor) Current Therapy in Theriogenology, 2nd ed. W.B. Sannders, Philadelphia.

Franklin,1986;Majeed and taha,1989;Thomas,1992;Nokes et al.,2009

Hindson, J.C. Turner, C.B. 1962. Observations on incomplete dilatation of the ovine cervix. Vet. Rec. 74:363

Ijaz, A. Talafha A.Q. 1999 Handbook of Veterinary obstetrics. Saunders, London.

Liggins, G.C. Fairclough, R.J. Grieves, S.A. Kendall, J.Z. Knox, S.J. 1973. 29, 111-114.

Majeed, A.F. 1994. Obstetrical problems and thier management in Iraqi goats. Small Rumin. Res. 14, 73-78.

Majeed, A.F. Taha, M.B Azawi, O. 1992. Caprine caesarean section. Small Rumin. Res.

9, 93-97.

Noakes et al.,2009

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Noakes, D.E. Parkinson, T.J. England, G.C.W, 2009 Arthuts Veterinary reproduction and obstetrics, 9th edition, DE Noakes, TJ Parkinson, GCW England (eds), Saunders, Edinburg, London.

Rahim and Majeed and Taha,1989;Noakes et al.,2009

Rahim, A.T Arthur, G.H, 1982. Obstetrical conditions in goats. Cornell Veterinarian, 72, 279-284.

Roke et al.,1990;Ghosh et al.,2004;Scott,2005

Scott, P.R. Naqvi, S.M. Pandey, G.K. Gautam. K.K. Joshi, A. Geethalakshmi. V. Mittal, J.P. 2005. The management and welfare of some common obive obstetrical problems in the United Kingdom. Vet. J. 170, 33-40. Review.

Thomas, J.O. 1992. Survey of the causes of dystocia in sheep. Vet. Rec. 127, 574-575.

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