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1 FIGURE S1: Liver histology with HBcAg staining. Immunoperoxidase staining was used to detect the presence of HBcAg in liver biopsy specimens from hepatitis B mono-infected and HIV-HBV co-infected patients.

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FIGURE S1: Liver histology with HBcAg staining. Immunoperoxidase staining was used to detect the presence of HBcAg in liver biopsy specimens from hepatitis B mono-infected and HIV-HBV co-infected patients.

A) Panel A shows the typical aspect of HBcAg immunohistochemical staining of a patient with chronic hepatitis B, with predominantly nuclear expression of HBcAg.

B) Panel B shows very strong and diffuse HBcAg expression with a mixed pattern (nuclear and cytoplasmic) in a patient co-infected with chronic hepatitis B virus and HIV.

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2

FIGURE S2: Core, but not surface, hepatitis B antigen was increased with HIV co-infection.

Liver biopsy specimens from HBV mono-infected and HIV-HBV co-infected patients were read and scored by a hepatic pathologist to determine prevalence of HBcAg and HBsAg by immunoperoxidase staining. Scores were based on a scale of 0-4, in which 0= none, 1+=<10%, 2+= 10-50%, 3+ = 50-90% and 4+=>90% of hepatocytes with positive antigen staining.

A) Distribution of HBcAg scores for HBV mono-infected and HIV-HBV co-infected groups.

Kruskal-Wallis test was used to compare the proportion of ordered categorical levels; p = 0.003.

0 10 20 30 40

HBV HBV-HIV

%

HBcAg

0 1 2 3 4

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B) Distribution of HBsAg scores in HBV versus HBV and HIV co-infected patients. Kruskal- Wallis test was used to compare the proportion of ordered categorical levels; p = 0.176

0 10 20 30 40 50 60

HBV HBV-HIV

%

HBsAg

0 1 2 3

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FIGUR counts.

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A) HBV infected as red. H

RE S3: HBV HBV DNA ed as part o

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els (x-axis) were designa s increased

els correlate erum were boratory tes

were graph ated as blue in HBV pat

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hed against H while HBV tients co-inf

ree of HBcA using quanti

HBcAg sco V-HIV co-in

fected with

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ores (y-axis) nfected patie

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g, but not CD . CD4 coun

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no- marked

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B) HBV DNA levels (x-axis) were plotted against CD4 counts (y-axis).

Only HBV-HIV co-infected patients had CD4 data. CD4 counts were independent of HBV DNA levels.

0 100 200 300 400 500 600 700 800 900 1000

CD4 Counts

3 4 5 6 7 8 9 10 11 12

DNA log

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6

C) CD4 counts were divided into 3 groups of <200, 200-500, and >500 (x-axis), and plotted versus HBV DNA level (y-axis). Distribution of HBV DNA (log) did not correlate with any CD4 group.

-1 0 1 2 3 4 5 6 7 8 9 10 11 12

<200 200-500 >500

CD4 Counts

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