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C. Treatment of cerebral edema should begin at recognition: administering hypertonic saline or

ENDOCRINE SYSTEM

C. Signs and Symptoms (Table 6.8)

3. C. Treatment of cerebral edema should begin at recognition: administering hypertonic saline or

mannitol, elevating the head of the bed, and per­

forming an emergent CT scan of the brain to evaluate degree of edema. Fluid administration should be decreased. If the Glascow Coma Scale score is less than 9, support of respiratory and neurologic status is necessary. Risk factors for cerebral edema include first presentation, high serum blood urea nitrogen (BUN) and CO2 ( reflective of the degree of dehydra­

tion and acidosis), administration of insulin bolus doses, bicarbonate administration, rapid glucose correction, aggressive fluid administration, and younger age at diagnosis.

Case Study 6.4

1. B. Hypothyroidism in the neonate is characterized by poor feeding, hypotonia, constipation, weight loss and decreased activity with weak cry. Lab results seen in hypothyroidism will reveal an ele­

vated TSH and low free T4. Treatment with levothy­

roxine should be initiated in conjunction with pediatric endocrinology as lack of treatment can lead to significant cognitive impairment.

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DEVELOPMENTAL ANATOMY OF THE GASTROINTESTINAL SYSTEM

A. Embryologic Development of the Digestive