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Amoebozoa

Dalam dokumen Immunity to Parasitic Infection (Halaman 74-79)

Introduction to Protozoan Infections

2.2 Amoebozoa

This supergroup consists of thirteen clades of amoebae and amoeboid flagel- lates. The Amoebozoa contains free-living, commensal, and parasitic species.

Free-living species inhabit both terrestrial and aquatic environments. Most move via directed cytoplasmic flow and extension pseudopodia, although a few groups utilise flagella during particular points in their life cycle. Organisms within this group also show a great deal of diversity in their organelle comple- ment. Some species have branching mitochondria with irregular cristae, de- generate or no mitochondria, or harbour a variety of endosymbionts. Several members of this group are parasites. of whichEntamoeba histolyticais the most important to human health.

2.2.1 Entamoebidae

This family is comprised of species that are commensals or parasites of the digestive systems of arthropods or vertebrates. Genera and species have

Table 2.1 There are 30 species of protozoa that commonly infect humans.Many of these species are commensals or opportunistic pathogens that only develop into medically important parasites in immunocompromised individuals. These species belong to five supergroups which are listed in underlined text, along with their mode of transmission, tissue tropism and global distribution. Species which were formally considered protozoa but are now known to be fungi are not listed.

Transmission Tissue tropism Distribution

Amoebozoa Archamoebae

Entamoeba histolytica environmental cysts or

direct transmission

small intestine worldwide

coli environmental cysts or

direct transmission

small intestine non-pathogenic

worldwide dispar environmental cysts or

direct transmission

small intestine non-pathogenic

worldwide

Endolimax nana environmental cysts cecum

non-pathogenic

worldwide

Iodamoeba b¨utschlii environmental cysts large intestine

non-pathogenic

worldwide Centramoebida

Acanthamoeba castellanii and other spp.

environmental cysts and trophozoites

respiratory and CNS worldwide Balamuthia mandrillaris environmental cysts and

trophozoites

respiratory and CNS North America Flabellinea

Sappinia diploidea environmental cysts and

trophozoites

respiratory and CNS North America Excavata

Metamonad Diplomonadida

Giardia lamblia environmental cysts or

direct transmission

small intestine worldwide Retortamonadida

Chilomastix mesnili environmental cysts cecum and colon

non-pathogenic

worldwide Trichomonadida

Trichomonas vaginalis direct transmission vagina, prostate, urethra and seminal vesicles

worldwide

tenax direct transmission oral cavity

non-pathogenic

worldwide

Pentatrichomonas hominis cecum and large

intestine non-pathogenic

worldwide

Dientamoeba fragilis unknown large intestine worldwide

(Continued)

Table 2.1 (Continued)

Transmission Tissue tropism Distribution Discoba

Kinetoplastida

Leishmania braziliensis Sandfly causes mucocutaneous

disease

Brazil

donovani sandfly causes visceral disease India and

China

infantum sandfly causes infantile visceral

Leishmaniasis

old world

major sandfly causes cutaneous

disease

old world

mexicana sandfly causes cutaneous

disease

new world

tropica sandfly causes cutaneous

disease

Middle East and India

Trypansoma cruzi reduviid bug systemic Central and

South America rangeli reduvid bug or faecal systemic

non-pathogenic

Central and South America

Trypanosoma brucei brucei systemic

non-pathogenic

Africa

b. gambiense systemic chronic

disease

Africa

b. rhodesiense systemic acute disease Africa

Heterolobosea

Naegleria fowleri environmental cysts and

trophozoites

respiratory and CNS worldwide Harosa

Aveolata Apicomplexa

Babesia microti and other

spp.

tick systemic generally

non-pathogenic

USA

Cystoisospora belli environmental oocytes small intestine worldwide

Cryptospordium parvum environmental oocysts small intestine worldwide

homis environmental oocysts small intestine worldwide

Plasmodium falciparum mosquito systemic worldwide

knowlesi mosquito systemic South East

Asia

malariae mosquito systemic worldwide

ovale mosquito systemic old world

vivax mosquito systemic worldwide

Toxoplasma gondii ingestion of

environmental oocysts or tissue bradyzoites

systemic worldwide

Table 2.1 (Continued)

Transmission Tissue tropism Distribution Ciliophora

Balantidium coli environmental cyst large intestine Common in the

Philippines, worldwide Stramenophiles

Blastocystis sp. environmental cyst intestine worldwide

Opisthokont Ichthyosporea

Rhinosporidium seeberi environmental nasal mucosa Asia

Archeaplastida Chlorellales

Prototheca wickerhamii environmental dermis worldwide

traditionally been differentiated based on nuclear morphology. The most im- portant species, Entamoeba histolytica, is the causative agent of amoebic dysentery and infects millions of people annually, causing at least 100,000 deaths per year. While it is primarily transmitted via contaminated water or food, sexual transmission is also common in certain communities.

2.2.2 Entamaoba histolytica

The life cycle of this parasite is relatively simple and comprised of five stages:

trophozoites, precysts; cyst; metacyst; metacystic trophozoites. Trophozoites inhabit the crypts of the large intestine and are highly motile, feeding on lu- minal content as well as mucus and material scavenged from the destruction of epithelial cells. While vacuoles containing digesting material are readily ap- parent in E. histolytica trophozoites, they have a number of unusual ultra- structural characteristics (Figure 2.2A and 2.2B). The cell is generally divided into two sections: a granular central mass called the endoplasm, that contains the nucleus; and a clear outer zone called the ectoplasm, that surrounds the endoplasm. The ectoplasm trails behind endoplasm as the protozoa moves.

The nuclei of these organisms have a prominent endosome at the centre, with achromatic fibrils radiating to the inner surface of the nuclear membrane.

Typical endoplasmic reticulum and Golgi apparatus are not readily apparent in these organisms, although recent studies indicate that they have mem- brane compartments that perform similar functions. They also lack conven- tional mitochondria, instead having structures called mitosomes or cryptons.

The function(s) of the mitosome is still unclear. Like conventional mitochon- dria, they possess a double membrane, and nuclear-encoded proteins are tar- geted to them with mitochondrial-like signal sequences. They do not have an organellular DNA, nor do they perform metabolic functions seen in aerobic or- ganisms such as oxidative phosphorylation. Currently, they are believed to be

Mitosome/crypton Ectoplasm Endoplasm

Endocytosed material in food vacuole

Nucleus

Nucleus

Karyosome Chromatoid Body

(A) (B)

(C) (D)(D)

cyst

trophozoite

Figure 2.2 Entamoeba histolyticabasic anatomy.(A) A phase contrast image of anE. histolyticatrophozoite. (B) The illustration shows an idealised image of a trophozoite and a cyst. The major organelles and other subcellular features are labelled. (C) A phase contrast image of anE. colicyst showing the characteristic multinucleate structure. (D) A transmission electron microscopy (TEM) image of the chromatoid body within theE. histolyticacyst.

degenerate mitochondria which play a role in Fe-S cluster assembly. However, additional metabolic functions will undoubtedly be identified.

Trophozoites multiply by binary fission. Under certain conditions, trophozoites invade the submucosa, causing inflammation and ulceration that spreads to the underlying muscularis mucosae and serosa. These ulcers can develop into serious lesions, via a combination of tissue destruction by the invading para- sites and immune reactions to amoebas and luminal bacteria carried into the wound. In the most extreme cases, there is perforation of the colon and dissem- ination of parasites to other organs, leading to life-threatening conditions.

In normal, asymptomatic infections trophozoites pass in stool formations to the lower sections of the colon. Dehydration of the stool stimulates them to condense into a precyst sphere that secretes a hyaline wall around itself. The cyst initially has a single nucleus, but these rapidly divide into four nuclei as the cyst matures (Figure 2.2B and 2.2C). Chromatoid bodies containing large amounts of RNA are prominent during the development of the cyst (Figure 2.2B and 2.2C). This quadrinucleate cyst (or metacyst) is then passed into the ex- ternal environment, where it can then infect a new host. After ingestion and

excysting in the small intestine, the cytoplasm and nuclei divide, forming eight small metacystic trophozoites.

Dalam dokumen Immunity to Parasitic Infection (Halaman 74-79)