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Cluster Headache

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Headache

4. HEADACHE TREATMENT

4.3. Cluster Headache

The intensity of each individual cluster headache attack is so severe that therapy must focus on prevention (see Box 6). In addition, most acute thera-pies will not become effective during the course of these brief attacks. Epi-sodic cluster headache is typically treated with preventive therapy for the ache Fig. 8. Therapeutic options for migraine, tension-type, and posttrauma headaches.

expected duration of the cluster cycle, usually approximately 6 weeks. Chronic cluster, which has no or only brief headache-free periods, is treated with daily, ongoing, preventive therapy. If patients are initially diagnosed when a cluster period has already reached peak severity, treatment with a short course of ste-roids is usually necessary. Plans should be made at that time, however, for initiation of preventive therapy at the start of the next cluster cycle.

Fig. 9. Choosing acute care medication.

Fig. 10. Choosing preventive therapy. Combining medication and alternative thera-pies maximizes treatment outcome.

Box 6

Treatment of Cluster Headache

• Episodic cluster (cluster duration ≥7 days, with pain-free period between clus-ters≥1 month)

 Preventive therapy: onset of cluster

 Discontinuation of nicotine and alcohol during cluster

 240–480 mg/day verapamil for 6 weeks

 2–8 mg/day methysergide for 6 weeks

 Preventive therapy: cluster at maximum intensity at time of treatment initiation

 Prednisone 10–60 mg/day for 1 week

 Rescue therapy

 6 mg subcutaneous sumatriptan

 100% O2 7 L/minute for 10 minutes by face mask

 Intranasal butorphanol

• Chronic cluster (cluster duration >1 year, with any pain-free periods during that year lasting <1 month)

 Preventive therapy

 Discontinuation of nicotine and alcohol

 240–480 mg/day verapamil

 250–1000mg/day valproic acid

 900–1800 mg/day gabapentin

 Rescue therapy

 100% O2 7 L/minute for 10 minutes by face mask Box 5

Choosing a Triptan

• Weigh needs for immediate relief against convenience/desirability of oral therapy

 Fastest relief from injectable or intranasal sumatriptan

 Patients typically prefer oral formulations

 Fast-acting oral triptans include rizatriptan, eletriptan, zolmitriptan, and sumatriptan

• Need for sustained relief

 Add nonsteroidal anti-inflammatory drugs to fast-acting triptan (25)

 Choose slower acting triptan

 Naratriptan and frovatriptan

• Desire for convenient formulation

 Orally disintegrating formulations (Maxalt MLT or Zomig ZMT)

• Sumatriptan nonresponders usually respond to alternative triptan.

 Only 19% of sumatriptan nonresponders fail zolmitriptan and rizatriptan (24)

ductive cycle in women. Estradiol is an important pain modulator, directly influencing neural function through a variety of neurotransmitters important for transmitting pain signals, including endorphins, serotonin, γ-aminobutyric acid (GABA), and dopamine (27). Generally, estradiol protects against pain, reducing pain perception as estradiol levels rise. Therefore, the pain threshold increases and headache frequency decreases for the majority of women during pregnancy (28,29). Conversely, when estradiol levels fluctuate or drop from high to low levels—as occurs with ovulation, menses, the placebo week of oral contraceptives, and after delivery—headache frequency increases. Because estradiol levels fluctuate during the perimenopausal period, headaches tend to worsen while other somatic symptoms of menopause—e.g., hot flashes—occur during early menopause. Headaches may also be aggravated during meno-pause by estrogen supplementation (30).

Headache management in women should focus on either minimizing changes in estradiol levels with estrogen supplementation when a decline in estradiol levels is expected or by pharmaceutically manipulating other important neurochemicals, such as using antidepressants or triptans to modulate serotonin levels, valproate or gabapentin to modulate GABA, or antinausea medi-cations to modulate dopamine (see Boxes 7 and 8) (31). Consideration should also be given to the risk for adverse effects of medications on the developing fetus when treating headaches during pregnancy (see Box 9). Most medications that can be used safely during pregnancy can be continued when breastfeeding.

Injectable sumatriptan, which is restricted during pregnancy, may be used when by a nursing mother if she pumps her milk and discards it for 4 hours after an injection and supplements the baby’s feeding with stored milk.

5. SUMMARY

Managing chronic headache begins with a reliable headache history to aid the health care provider in identifying common headache patterns. Worrisome headaches are generally associated with new headache patterns, pain in the back of the head or neck, aging (>50 years old), or abnormal neurological ex-amination findings (21). A diary that is used to record both headache activity and medication use can be helpful to the clinician in correctly identifying

head-Box 7

Treating Menstrual Headache: Perimenstrual Prevention

• Hormone therapy

 7-day 100-mg estrogen patch

 Eliminate placebo week from oral contraceptives for 2 or 3 months

• Acute-care medications

 Nonsteroidal anti-inflammatory drugs (excluding aspirin or ibuprofen)

 2.5 mg naratriptan twice daily

 2.5 mg frovatriptan once or twice daily

• Preventive medications

 β-blocker

 Antidepressant (excluding fluoxetine)

 Calcium channel blocker

 Antiepilepsy drug (valproic acid or gabapentin)

All medications should be used at usual dose for 3 days before the expected menstrual period and during first 2–4 days of menses. Do not use unless diary confirms headache occurrence exclusively in association with menses.

Box 8

Treating Headache During Menopause

• Determine if there has been a change in headache pattern notable enough to warrant additional evaluation

• Adjust estrogen replacement therapy if it aggravates headache

 Use noncycling, transdermal route

 Reduce estrogen dose

 Change estrogen-replacement product

• Add standard headache-preventive therapy in conjunction with estrogen replacement

patterns, the contribution of medication overuse to headache, and the relation-ship of headache to menstruation. Identification and elimination of medication overuse is the first step in successful headache management. Choice of head-ache therapy depends on headhead-ache frequency and disability. Combining medi-cation and nonmedimedi-cation therapies maximizes treatment outcome.

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 Medications

 β-blocker

 Selective serotonin reuptake inhibitor antidepressant

 Bupropion

 Gabapentin (in early pregnancy; stop in third trimester)

 Nonmedication therapy

 Relaxation and biofeedback

 Stress management

 Discontinuation of nicotine and caffeine

 Regular meals and sleep

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2. Features associated with potentially serious headache that requires additional evaluation include:

a. Posterior head or neck pain

b. Pain beginning after the age of 50 years c. Change in headache pattern

d. Abnormal neurological examination e. All of the above

3. The most common type of chronic headache seen in the primary care office is:

a. Migraine b. Tension-type c. Posttraumatic d. Cluster

4. Which of the following headaches is experienced as short pain episodes, typically lasting

<2 hours?

a. Migraine b. Tension-type c. Posttraumatic d. Cluster

e. Medication overuse

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From: Chronic Pain: A Primary Care Guide to Practical Management Edited by: D. A. Marcus © Humana Press, Totowa, NJ

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