• Tidak ada hasil yang ditemukan

Hubungan Helicobacter Pylori Cytotoxin-Associated Gene A (CagA) Positif Dan Negatif Dengan Derajat Keparahan Gastritis Berdasarkan Histopatologi

N/A
N/A
Protected

Academic year: 2017

Membagikan "Hubungan Helicobacter Pylori Cytotoxin-Associated Gene A (CagA) Positif Dan Negatif Dengan Derajat Keparahan Gastritis Berdasarkan Histopatologi"

Copied!
7
0
0

Teks penuh

(1)

DAFTAR PUSTAKA

1. El-Zimaity HMT. Recent advances in the histopathology of gastritis. Current Diagnostic Pathology. 2007;13:340-8.

2. Djojoningrat D. Dispepsia Fungsional. Dalam: Sudoyo AW, editor. Buku ajar Ilmu Penyakit Dalam. Jilid I edisi IV. Jakarta: BP FKUI; 2006: 354-6.

3. Rugge M, Genta RM. Staging and grading of chronic gastritis. Human pathology. 2005;36:228-33.

4. Hirlan, Gastritis dalam Buku Ajar Ilmu Penyakit Dalam Edisi 5, Internal Publishing : 2009 ; 509-512

5. Pratomo BW. Gastritis dan gastropati dalam buku ajar gastroenterologi. Edisi I. Internal publishing. 2011:307-311.

6. Mayo clinic. Gastritis.2014 [diunduh 20 Maret 2015]. Tersedia dari : http://www.mayoclinic.org

7. Rugge M, Pennelli G, Pilozzi E, Fassan M, Ingravallo G, Russo VM, et al. Gastritis: the histology report. Digestive and Liver Disease. 2011; 43S:373– 84.

8. Adibi P. Gastritis. [Diunduh 19 Mei 2015]. Tersedia dari: http://med.mui.ac.ir 9. Szoke D. Genetic factors related to the histological and macroscopic lesions of

the stomach. [disertasi]. Budapest: Semmelweis University; 2009: 61-7. 10.Toljamo K. Gastric erosions – clinical significance and pathology. A

long-term follow-up study. University of Oulu, Finland:Acta Univ Oul; 2012:80-1. 11.Zakaria Z. The role of interleukin-10 in Helicobacter pylori infection. Master

[Tesis]. Nottingham,UK: University of Nottingham; 2010: 55.

12.Fox JG, Megraud F. Helicobacter. In: Murray PR, editor. Manual of clinical microbiology. 9th ed. Pensylvania: Elsevier Mosby; 2007 : 947-62.

(2)

14.Banerjee A, Mukhopadhyay AK, Paul S, Bhattacharyya A and Swarnakar S. Unveiling the Intricacies of Helicobacter pylori-induced Gastric Inflammation: T Helper cells and Matrix Metalloproteinases at a Crossroad. In: Mozsik G, editor. Current Topics in Gastritis. Croatia: InTech Publishers; 2013: Chapter 7.

15.Hindawi Publishing Corporation BioMed Research International Volume 2014, Article ID 142980, 7 pages [diunduh 20 Februari 2016] .Tersedia dari: http://dx.doi.org/10.1155/2014/142980

16.Rehnberg-Laiho L, Rautelin H, Koskela P, Sarna S, Pukkala E, Aromaa A, et al. Decreasing prevalence of helicobacter antibodies in Finland, with reference to the decreasing incidence of gastric cancer. Epidemiol Infect. 2001;126:42-37.

17.Matsukura N, Yamada S, Kato S, Tomtitchong P, Tajiri T, Miki M, et al. Genetic differences in interleukin-1 betapolymorphisms among four Asian populations: an analysis of the Asian paradox between H. pylori infection and gastric cancer incidence. J Exp Clin Cancer Res. 2003; 22: 55-47.

18.Adam B, et al. Cellular immune activation and clinical manifestation of funcional dyspepsia. Z Gastroenterol 2008;26-46.

19.Elseweidy MM. Helicobacter pylori ifection and its relevant to chronic gastritis. INTECH.2013.http://dx.doi.org/105772/46056

20.Conteduca vincenza, domenico sansonno, gianfranco lauletta, sabino russi, giuseppe ingravallo and franco dammacco. H. pylori infection and gastric cancer: State of the art. international journal of oncology. 2013.42: 5-18. 21.D W, Chey, C B, Wong. American college of gastroenterology guidline on the

management of H.pylori infetion. Am J Gastroenterol 2007;102:1808-1825 22.Lew E. Peptic ulcer disease. In: Greenberger NJ, editor. Current Diagnosis and

Treatment Gastroenterology, Hepatology, and Endoscopy. New York: McGraw Hill; 2009:175-83.

(3)

24.Fornai M, Antonioli L, Colucci R, Tuccori M, Blandizzi C. Pathophysiology of gastric ulcer development and healing: molecular mechanism and novel therapeutic options. In: Chai J, editor. Peptic Ulcer Disease. Shanghai: Intech; 2011: 113-42.

25.Jones MK, Tomikawa M, Mohajer B, Tarnawski AS. Gastrointestinal mucosal regeneration: role of growth factors. Frontiers in Bioscience. 1999b;4:303-9. 26.Brzozowski T, Konturek PC, Konturek SJ, Brzozowska I, Pawlik T. Role of

prostaglandins in gastroprotection and gastric adaptation. Journal of physiology and pharmacology. 2005; 56:33-55.

27.Tarnawski AS, Chai J, Jones MK. Esophageal and Gastrointestinal Microcirculation: Essential for Mucosal Protection, a Target for Injury, and a Critical Component of Injury and Ulcer Healing. In: Ishii H, Suematsu M, Tanishita K, editor. Organ microcirculation: a gateway to diagnostic and therapeutic intervention. Tokyo: Springer; 2005:49-61.

28.Mueller A, Merrel DS, Grimm J, Falkow S. Profiling of microdissected gastric epithelial cells reveals a cell-type specific response to Helicobacter pylori infection. Gastroenterology. 2004;127:1446–62.

29.Markus S, Ruggiero P, Rappuoli R, Bagnoli F et al. Helicobacter pylori CagA: from pathogenic mechanisms to its use as an anti-cancer vaccine. Frontier in immunology. doi: 10.3389/fimmu.2013.00328

30.Akopyants NS, Clifton SW, Kersulyte D et al. Analyses of the cag pathogenicity island of Helicobacter pylori. Mol Microbiol 1998; 28: 3753 31.Kuipers EJ, Perez-Perez GI, Meuwissen SG, Blaser MJ. Helicobacter pylori

and atrophic gastritis: importance of the cagA status. J Natl Cancer Inst 1995;

87: 1777–80.

32. Crabtree JE, Taylor JD, Wyatt JI et al. Mucosal IgA recognition of Helicobacter pylori 120 kDa protein, peptic ulceration, and gastric pathology.

Lancet 1991; 338: 332–5.

33.Blaser MJ, Perez-Perez GI, Kleanthous H et al. Infection with Helicobacter pylori strains possessing cagA is associated with an increased risk of

(4)

34.Parsonnet J, Friedman GD, Orentreich N, Vogelman H. Risk for gastric cancer in people with CagA positive or CagA negative Helicobacter pylori infection.

Gut 1997; 40: 297–301.

35.Rugge M, Busatto G, Cassaro M et al. Patients younger than 40 years with gastric carcinoma – Helicobacter pylori genotype and associated gastritis

phenotype. Cancer 1999; 85: 2506–11.

36.Shimoyama T, Fukuda S, Tanaka M, Mikami T, Munakata A, Crabtree JE. CagA seropositivity associated with development of gastric cancer in a

Japanese population. J Clin Pathol 1998; 51: 225–8.

37.Nomura M, Lee J, Stemmermann GN, Nomura RY, Perez-Perez GI, Blaser MJ. Helicobacter pylori CagA seropositivity and gastric carcinoma risk in a

Japanese American population. J Infect Dis 2002; 186: 1138–44.

38.Kikuchi S, Crabtree JE, Forman D, Kurosawa M. Association between infections with CagA-positive or -negative strains of Helicobacter pylori and

risk for gastric cancer in young adults. Am J Gastroenterol 1999; 94: 34559. 39.Segal ED, Cha J, Lo J, Falkow S, Tompkins LS. Altered states: Involvement

of phosphorylated CagA in the induction of host cellular growth changes by

Helicobacter pylori. Proc Natl Acad Sci USA 1999; 96: 14 55964.

40.Asahi M, Azuma T, Ito S et al. Helicobacter pylori CagA protein can be tyrosine phosphorylated in gastric epithelial cells. J Exp Med 2000; 191: 593– 602.

41.Stein M, Rappuoli R, Covacci A. Tyrosine phosphorylation of the Helicobacter pylori CagA antigen after cag-driven host cell translocation.

Proc Natl Acad Sci USA 2000; 97: 1263–8.

42.Odenbreit S, Puls J, Sedlmaier B, Gerland E, Fischer W, Haas R.Translocation of Helicobacter pylori CagA into gastric epithelial cells by type IV secretion.

Science 2000; 287: 1497–500.

43.Backert S, Ziska E, Brinkmann V et al. Translocation of the Helicobacter pylori CagA protein in gastric epithelial cells by a type IV secretion apparatus.

(5)

44.Selbach M, Moese S, Hauck CR, Meyer TF, Backert S. Src is the kinase of the Helicobacter pylori CagA protein in vitro and in vivo. J Biol Chem 2002; 277: 677-758.

45.Stein M, Bagnoli F, Halenbeck R, Rappuoli R, Fantl WJ, Covacci A. c- Src/Lyn kinases activate Helicobacter pylori CagA through tyrosine

phosphorylation of the EPIYA motifs. Mol Microbiol 2002; 43: 971–80 46.orrea P, Haenszel W, Cuello C, Tannenbaum S, Archer M. A model for gastric

cancer epidemiology. Lancet 1975; 2: 58–60.

47.Yamazaki S, Yamakawa A, Ito Y et al. The CagA protein of Helicobacter pylori is translocated into epithelial cells and binds to SHP- 2 in human gastric

mucosa. J Infect Dis 2003; 187: 3347

48. R. Schindler, Gastritis,William Heinmann, London, UK, 1947.

49. Marshall BJ, Warren JR, “Unidentified curved bacilli in the stomach of patients with gastritis and peptic ulceration,”The Lancet, vol. 1, no. 8390, pp. 1311–1315, 1984.

50. Jacobson BC, Crawford JM, Farraye FM, “GI tract endoscopic and tissue processing techniques and normal histology in surgical pathology of the GI tract,” in Liver, Biliary Tract and Pancreas, R. D. Odze and J. R. Goldblum, Eds., Elsevier,2009.

51.Carneiro F, Fonseca E, Simoes S, “Epithelialdegeneration induced by Helicobacter pylori,” Human Pathology, vol. 23, no. 11,1992, p. 1314–1315. 52. Leung KM, Hui Pk, Chan W, Thomas T, “Helicobacter pylori-related gastritis

and gastric ulcer: a continuum of progressive epithelial degeneration,” American Journal of Clinical Pathology, vol. 98, no. 6,1992, p. 569–574. 53.Genta RM, Lew GW, Graham DY, “Changes in the gastric mucosa following

eradication of Helicobacter pylori,” Modern Pathology, 1993,vol. 6; no. 3, p. 281–289

(6)

55.Sapari NS, Loh M, Vaithilingam A, Soong R, “Clinical potential of DNA methylation in gastric cancer: a metaanalysis,” PLoS One, vol. 7, no. 4,Article ID e36275, 2012.

56.Carrasco G, Diaz J, Valbuena JR, et al., “Overexpression of p73 as a tissue marker for high-risk gastritis,” Clinical Cancer Research, vol. 16, no. 12, pp. 3253–3259, 2010

57. Dixon MF, Genta RM, “Classification and grading of gastritis. The update Sydney System. International workshop on the Histopathology of Gastrits, Houston 1994” Am J Surg Pathol β0(10), 1161-1181, 1996

58. Elseweidy MM, Taha MM, younis NN et al, “ Gastritis induced by helicobacter pylori infection in experimental rats” Dig Dis Sci 55, β770 -2777,2010

59.Rugge M, Pennelli G, Pilozzi E, Fassan M, et al. Gastritis: The histology report. Digestive and liver Disease 2011;43:373-84

60.Yulida E, Oktaviynti IK, Rosida L. Gambaran derajat infiltrasi sel radang dan infeksi Helicobacter pylori pada biopsi lambung pasien gastritis. Berkala Kedokt. 2013;(1): 47-58

61.Szoke D. Genetic factors related to the histological and macroscopic lesions of the stomach [Disertasi]. Budapest: Semmelweis University; 2009.pp.7-61 62.Aydin O, Egilmez R, Karabacak T, Kanik A. Interobserver variation in

Histophological assesment of Helicobacter pylori gastritis. World J Gastroenterol 2003;9:2232-5

63.Rugge M, Genta RM. Staging and grading of chronic gastritis. Human Pathol.2005;36:228-33

64.Rugge M, Correa P, Di Mario F, El-Omar E, Fiocca R, Geboes K, et al. OLGA staging for gastritis: A tutorial. Dig liver Dis.2008;40(8):650-8.doi:10.1016/j.dld.2008.02.030.

(7)

Referensi

Dokumen terkait

Adapun kisi-kisi instrumen yang digunakan dalam penelitian ini antara lain:.. 33 Arikunto , Prosedur Penelitian Suatu Pendekatan Praktik,

Selama ini tanpa disadari, siswa telah belajar dengan lebih banyak menerima materi pelajaran, namun tidak pernah diajarkan bagaimana cara belajar yang baik.

menyatakan bahwa pengaruh pemahaman konsep dan motivasi belajar terhadap hasil belajar materi trigonometri secara bersama-sama adalah 44,1% dan 55,9% dipengaruhi

Berdasarkan hasil observasi dan wawancara pada pihak manager Ayam Penyet Surabaya Medan kedua variabel bebas yang diteliti yaitu variabel kualitas pelayanan dan kualitas

PETUNJUK BELAJAR: Sambil membaca ulasan ini ingatlah kembali tentang nutrisi vitamin dan asam lemak esensial dari matakuliah Dasar Nutrisi Ternak dan BMT.

Low Pass Filter (LPF) adalah filter yang hanya melewatkan sinyal dengan frekuensi yang lebih rendah dari frekuensi cut-off ( f c ) dan akan melemahkan sinyal.. dengan frekuensi

berhubungan dengan pembukaan rahasia nasabah bank oleh OJK.. dalam hal pemeriksaan perpajakan melalui aplikasi elektronik. berdasarkan POJK No.25/pojk.03/2015.

Using e-portfolios, students also find it easier to track the progress they have achieved. Thus they are easier to plan appropriate way to study according to their shortcomings